• Possible link between depression and HHV-6 infection in childhood

    Serologic studies of several herpesviruses find that only HHV-6 may be linked to depression.

  • Reactivation of latent herpesviruses and HERVS after mild COVID-19

    Findings are accentuated in people with myalgic encephalomyelitis/chronic fatigue syndrome.

  • Virus specific T cell infusions show promise for preventing HHV-6B reactivation in transplant patients

    Review highlights the importance of cell mediated immunity and the limitations of antivirals, vaccines and immunoglobulin therapy for HHV-6, VZV and HSV.

  • Artesunate compares favorably to herpesvirus antivirals in suppressing HHV-6 and HHV-7

    Non-randomized study finds somewhat more potent reduction of virus in leucocytes over several months of treatment.

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View Post

Short-course foscarnet at an early stage may suppress HHV-6 reactivation following allogenic HCT

Observational study suggests that an early one-week treatment at the first sign of viral reactivation may achieve clinical benefits and avoid antiviral toxicity.

View Post

COVID-19 patients with HHV-6 DNA and CNS symptoms showed increased expression of miR-155

Both HHV-6 and specific microRNAs may correlate with neurological symptoms in COVID-19.

View Post

Accurate Multiplex Assay for Viral Causes of Vesicular Diseases

Assay for monkeypox virus, HSV1-2, HHV-6, VZV and enteroviruses.

View Post

Herpesvirus Reactivation Common in Severe COVID-19

HHV-6 was the most common herpesvirus found when tested in nasal swabs, but EBV was the most common in plasma.

View Post

Systematic review of pityriasis rosea following COVID-19 and/or vaccination

Pityriasis rosea was reported more frequently after vaccination than during or after acute infection.

View Post

HHV-6 DNA load in whole blood and elevated EBNA antibodies predict progression in first clinical episodes of demyelination in multiple sclerosis

Patients with viral loads suggestive of iciHHV-6 had the greatest risk of developing demyelinating disease.

View Post

Systematic review: HHV-6 causes 77% of encephalitis cases in hematopoietic cell transplant patients, with a high mortality rate

1.1% of 72,423 HCT patients developed encephalitis with an all-cause mortality rate of 60%.

View Post

Differentiating incidental from disease-causing HHV-6 infections in pediatric spinal fluid samples

Clinical assessment revealed that 91% of iciHHV-6 cases and 33% of non-iciHHV-6 cases were incidental.

View Post

BioFire assay identifies HHV-6 as the most frequent cause of meningoencephalitis in infants 4-11 months

All cases found to be iciHHV-6 were assumed to be false positives.

View Post

High levels of HHV-6A DNA found in blood of 52% of kidney transplant patients

HHV-6A was the most common virus identified, but was not linked to worse outcomes compared to other viral infections.

View Post

A tribute to Dharam Ablashi

The HHV-6 field has lost a pioneer.

View Post

DRESS, a syndrome linked to viral reactivation, may be triggered by several antivirals

Ribavarin, ritonavir, abacavir and lamivudine were the primary triggers.

View Post

Small study finds relatively high foscarnet concentrations in CSF in HHV-6 encephalitis

CSF foscarnet concentrati.ons were very near IC50 and were followed by sharp reductions in viral load.

View Post

Single cell transcriptomics of iciHHV-6 cell lines immortalized by EBV infection

Transcription of HHV-6 genes was rare, but occurred most often in cells with the highest levels of EBV transcription.

View Post

Are herpesviruses reactivated more often in acute COVID-19?

Systematic review and meta-analysis found strong evidence for EBV in the seriously ill; only 6 of 36 studies included HHV-6 testing.

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ABOUT THE HHV-6 FOUNDATION

The HHV-6 Foundation in a non-profit entity founded to encourage scientific exchange between investigators and to provide pilot grants for promising scientific and clinical research on the under- appreciated viruses HHV-6A and HHV-6B.

The Foundation sponsors international conferences and supports scientists and clinicians seeking to clarify the role of the two HHV-6 viruses in disease. Since HHV-6A and HHV-6B can smolder in the brain and other organs without circulating in the peripheral blood or plasma, identifying chronic infection is a challenge.

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