• 2019 Conference

    The 11th International Conference on HHV-6 & 7 will be held in beautiful Quebec City, Canada June 23–26, 2019.

  • HHV-6 in the news!

    After years of very little interest by the scientific community, there has suddenly been a lot of interest in HHV-6A, which along with HHV-7, appears to be central to the progression of Alzheimer’s disease.

  • Gene-expression network analysis points to HHV-6A as a key driver in upregulating genes leading to Alzheimer’s disease progression

    Investigators at Mt Sinai used “big data” models to determine that the genes involved with fighting Alzheimer’s are the same ones that fight virus. They found HHV-6A and HHV-7 to be more abundant in Alzheimer’s brains, and singled out HHV6-A as a key modulator of the genes involved in amyloidosis and neuronal death.

  • HHV-6 and HSV1 dramatically accelerate amyloid plaque production in Alzheimer’s model

    Researchers at Harvard studied how neurons responded to the presence of herpesviruses HSV1 and HHV-6, and found that they rapidly induce amyloid plaque production within 24 to 48 hours.

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Latest Scientific News Articles

TNF-α predicts HHV-6 reactivation and disease progression in patients with DIHS/DRESS

In All, Drug Hypersensitivity, Latest Scientific News by hhv6foundationFebruary 28, 2014

While elevated TNF-a detected at the early onset of disease is a strong indicator for the early recognition of HHV-6 reactivation, this finding also reflects important therapeutic information that could be used as an early diagnostic marker of DIHS/DRESS.

Spotlight on Horvat & Reynaud from INSERM

In All, Animal Models, Latest Scientific News by hhv6foundationFebruary 28, 2014

Branka Horvat, MD, PhD, Director of Research at the International Centre for Infectiology Research in Lyon, France (INSERM), and her doctoral student Josephine Reynaud have recently published an important paper on a new transgenic mouse model

Activation of ciHHV-6 by telomeric circle formation

In All, ciHHV-6, Latest Scientific News by hhv6foundationJanuary 29, 2014

Additional evidence that shortened telomeres can cause the release and possible activation of integrated HHV-6.

Measles immunosuppression may cause abortive, subclinical reactivation of HHV-6

In All, Latest Scientific News by hhv6foundationJanuary 29, 2014

Dr. Yoshizo Asano and colleagues in Japan report that in measles immunosuppression, cells sensitized to HHV-6B immediate early and early antigen proteins may suppress replication cycles and virus formation, resulting in an “incomplete” subclinical state of reactivation.

New HHV-6 reagent available from The HHV-6 Foundation: DR7 Mab

In All, Latest Scientific News by hhv6foundationJanuary 29, 2014

We are pleased to announce that with the help of a few HHV-6 specialists, especially Prof. Louis Flamand from Canada, we have generated the monoclonal antibody DR-7.

REVIEW: Important differences between the HHV-6A and HHV-6B viruses

In All, Epidemiology, Latest Scientific News by hhv6foundationDecember 22, 2013

An international group of experts summarize the significant differences between the two viruses.

Human herpesvirus replication is activated by apoptosis

In All, Latest Scientific News by hhv6foundationOctober 31, 2013

A group led by Steven Zeichner has published evidence that suggests the replication of all human herpesviruses can be activated by apoptosis.

Humanized mouse model demonstrates HHV-6A infection can cause significant immune dysfunction

In All, Animal Models, Latest Scientific News by hhv6foundationOctober 31, 2013

Could treatment of HHV-6A co-infections slow AIDS progression?

MCP-1 found elevated in the CSF of patients with HHV-6B encephalopathy

In All, Encephalitis & Encephalopathy, Latest Scientific News by hhv6foundationOctober 31, 2013

Japanese group identifies several cytokine and chemokine responses associated with HHV-6B encephalopathy.

HHV-6B infection promotes unique pro-inflammatory effects tied to neurocognitive decline and artherosclerosis

In All, Cognitive Dysfunction, Latest Scientific News by hhv6foundationSeptember 30, 2013

HHV-6A and HHV-6B each express distinct chemokines that are uniquely capable of activating key inflammatory cytokines.

New HHV-6B specific cellular receptor identified

In All, Latest Scientific News by hhv6foundationAugust 30, 2013

CD134, a member of the TNF receptor superfamily, has now been shown as a specific entry receptor for HHV-6B.

Chlamydia infection induces activation of ciHHV-6 leading to linear and circular DNA

In All, ciHHV-6, Latest Scientific News by hhv6foundationMay 6, 2013

A laboratory at the University of Wurzburg has published evidence that Chlamydia trachomatis may induce the activation of ciHHV-6.

Why does HHV-6 reactivate preferentially in CBT?

In All, Latest Scientific News by hhv6foundationJanuary 31, 2013

A group from France has reported new findings which may help explain the mystery behind the increased rates of HHV-6 reactivation and encephalitis observed among umbilical cord blood transplant (CBT) patients.

Bortezomib may increase risk of HHV-6 reactivation in myeloma patients

In All, Latest Scientific News by hhv6foundationJanuary 31, 2013

A new study has reported that the use of Bortezomib, known commercially as Velcade, may increase the risk of HHV-6 reactivation in myeloma patients undergoing stem cell transplantation.

HHV-6 induces oxidative stress and chlamydial persistence

In All, Latest Scientific News by hhv6foundationNovember 16, 2012

Findings indicate that HHV-6 infection leads to decreased glutathione production, increased oxidative stress, and induces chlamydial persistence during co-infection

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ABOUT THE HHV-6 FOUNDATION

The HHV-6 Foundation in a non-profit entity founded to encourage scientific exchange between investigators and to provide pilot grants for promising scientific and clinical research on the under- appreciated viruses HHV-6A and HHV-6B.

The Foundation sponsors international conferences and supports scientists and clinicians seeking to clarify the role of the two HHV-6 viruses in disease. Since HHV-6A and HHV-6B can smolder in the brain and other organs without circulating in the peripheral blood or plasma, identifying chronic infection is a challenge.

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